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Are you suffering from folic acid deficiency? I have good news that would help you to reduce your risk of heart disease, stroke, coronary artery disease, cancer, depression, memory loss, Alzheimer’s disease, venous occlusion of the retina, and more with a single form, a highly absorbable folic acid called L-methyl folate.

Increased level of homocysteine may result in endothelial dysfunction by oxidative damage. Research has shown that Hyperhomocysteinemia is the cause of venous or arterial thrombosis.

If you have had a high level of homocysteine in the past, you may want to check polymorphism for your blood or gene defect that reduces your capability to utilize folic acid. The test is known DNA analysis (MTHFR) and checks gene mutations. These mutations and their associated risks are inherited. So if the result is positive, then family members are also considered to be at-risk.

The MTHFR enzyme is responsible for creating the circulating form of folate. Folate is essential for regulating our levels of homocysteine. A normal homocysteine level must be less than 10. The defects in this enzyme can lead to elevated homocysteine levels. High serum levels of homocysteine have been associated with an increased risk of stroke, coronary artery disease, myocardial infarction and venous thrombosis. In women, there have been complications during pregnancy and an increased risk of fetal open neural tube defects.

On vascular disease, these polymorphisms may increase the risk of vascular disease and all the disorders mentioned above. L-methylfolate plays a key role in maintaining the stability of the gene. Much evidence suggests that an inadequate supply of folic acid in the diet can be a risk factor for several human diseases, including Alzheimer’s disease, Down syndrome cardiovascular disorders, and neonatal malformations.

On cancer, in conditions of deficiency of folic acid, research has shown that mutated gene is associated with high risk of cancer at several sites including breast, colon, cervical, prostate and gastric but L- methyl folate supplement can help in such condition.

Concerning depression, deficiency of folic acid may elevate the risk of depression and decrease the activities of antidepressants. People with an inherited polymorphism have a reduced formation of folic acid which may lead to a potential folate deficiency and major depression. When antidepressants are augmented with L-methyl folate, a more absorbable form of folic acid, the results have been recorded to be better.

L-methylfolate acts as an essential controller of a vital co-factor in the synthesis of neurotransmitters. By increasing the synthesis of neurotransmitters, L-methylfolate is capable of increasing the antidepressant actions of known antidepressants.

Even if you have the mutated gene polymorphism or, you can make all of the above diseases by adopting an extremely absorbable form of L-methyl folate. Hyperhomocysteinemia is a modifiable risk factor for the thrombotic disease. It has been observed that those with elevated homocysteine levels benefit the most from supplementation with L-methylfolate.

The conclusion is that folic acid deficiency is easily corrected, and the many health advantages of using a folic acid supplement like L-methylfolate make the correction of this imbalance an easy decision.

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